Research Use Only. KIRhub outputs are computational research artifacts. They are not validated for clinical decision-making, diagnosis, or treatment.

Primary targets: CDK4_CYCLIN_D1, CDK4_CYCLIN_D3, CDK4_CYCLIN_D2, CDK6_CYCLIN_D1, CDK6_CYCLIN_D3, CDK6_CYCLIN_D2 · FDA status: FDA Approved

Selectivity scorecard

MeasuredDerived
KISS
98.75
Gini
0.673
CATDS
0.020

Computed from wild-type kinome inhibition at 1 μM. Gini reproduces the published values within tolerance; KISS and CATDS are computed but pending reconciliation with the paper's reference code.

Polypharmacology radar

MeasuredDerived

Top 20 strongest-inhibited wild-type kinases for Palbociclib. Strongest target: CDK4_CYCLIN_D3 at 99.6% inhibition.

Accessible data table
RankTargetInhibition %Residual activity %
1CDK4_CYCLIN_D399.6%0.4%
2CDK4_CYCLIN_D199.0%1.0%
3CDK6_CYCLIN_D198.6%1.4%
4CDK6_CYCLIN_D398.5%1.5%
5CLK191.4%8.6%
6CLK485.9%14.1%
7CDK9_CYCLIN_T284.4%15.6%
8ULK280.1%19.9%
9STK1678.1%21.9%
10DYRK1B77.1%22.9%
11PKCNU_PRKD372.8%27.2%
12PKCMU_PRKD170.7%29.3%
13CDK2_CYCLIN_O65.7%34.3%
14EIF2AK265.6%34.4%
15CAMK2D65.0%35.0%
16TRKC64.4%35.6%
17CLK262.8%37.2%
18CDK9_CYCLIN_K62.5%37.5%
19PKMYT160.5%39.5%
20DYRK358.8%41.2%

Selectivity landscape

MeasuredDerived

Where Palbociclib sits in the 92-drug selectivity landscape (KISS vs Gini). The highlighted point is Palbociclib.

Atlas insights for Palbociclib

MeasuredReference

Pathway-space view of what this drug actually does, drawn from the Pathway Atlas.

On-target vs off-target shadow

DerivedMeasured

How much of this drug's pathway perturbation comes from primary targets vs polypharmacology vs 2nd-order propagation. When off-target dominates, the FDA label is the smallest description of the drug.

On-target0%
Off-target100%
Ghost (2nd-order)0%
PathwayCompositionTotal |Π|
ADIPOGENESIS
650.74
ALLOGRAFT_REJECTION
2037.71
ANDROGEN_RESPONSE
941.95
ANGIOGENESIS
299.76
APICAL_JUNCTION
2170.44
APICAL_SURFACE
167.55
APOPTOSIS
2798.26
BILE_ACID_METABOLISM
393.97
CHOLESTEROL_HOMEOSTASIS
376.19
COAGULATION
138.23
COMPLEMENT
1237.60
DNA_REPAIR
973.64
E2F_TARGETS
3567.25
EPITHELIAL_MESENCHYMAL_TRANSITION
1042.28
ESTROGEN_RESPONSE_EARLY
1711.20
ESTROGEN_RESPONSE_LATE
1899.13
FATTY_ACID_METABOLISM
223.74
G2M_CHECKPOINT
3223.91
GLYCOLYSIS
1317.26
HEDGEHOG_SIGNALING
420.15
HEME_METABOLISM
780.53
HYPOXIA
1873.32
IL2_STAT5_SIGNALING
861.51
IL6_JAK_STAT3_SIGNALING
1400.17
INFLAMMATORY_RESPONSE
1224.99
INTERFERON_ALPHA_RESPONSE
170.66
INTERFERON_GAMMA_RESPONSE
1870.67
KRAS_SIGNALING_DN
654.77
KRAS_SIGNALING_UP
938.52
MITOTIC_SPINDLE
2540.17
MTORC1_SIGNALING
1549.27
MYC_TARGETS_V1
1997.06
MYC_TARGETS_V2
519.51
MYOGENESIS
1435.80
NOTCH_SIGNALING
283.37
OXIDATIVE_PHOSPHORYLATION
349.75
P53_PATHWAY
1587.49
PANCREAS_BETA_CELLS
332.32
PEROXISOME
541.07
PI3K_AKT_MTOR_SIGNALING
2891.61
PROTEIN_SECRETION
673.45
REACTIVE_OXYGEN_SPECIES_PATHWAY
158.76
SPERMATOGENESIS
816.14
TGF_BETA_SIGNALING
935.19
TNFA_SIGNALING_VIA_NFKB
1886.04
UNFOLDED_PROTEIN_RESPONSE
870.11
UV_RESPONSE_DN
1715.01
UV_RESPONSE_UP
1449.49
WNT_BETA_CATENIN_SIGNALING
1314.34
XENOBIOTIC_METABOLISM
657.13

See this drug on the perturbation map →

Hallmarks-of-Cancer reach

DerivedReference

Projection onto the 10 canonical Hanahan & Weinberg hallmarks. Breadth = how many hallmarks this drug meaningfully perturbs.

ProliferationEvading apoptosisAngiogenesisInvasion / metastasisReplicative immortalityDeregulated metabolismImmune evasionGenome instabilityInflammationGrowth signaling

Breadth = 3.15 bits (max possible across 10 hallmarks = 3.32 bits). Multi-hallmark agent — broad polypharmacology.

Compare against the full catalog →

Anti-tumor matches — the "ideal patient" search

ModeledDerived

Top 5 real tumors closest to this drug's ideal patient (the tumor whose pathway state = −Π_d). Closest match cosine = 0.872

SampleCancer typecos to ideal
EPT0291EPN0.872
TCGA-CF-A5U8-01A-11R-A28M-070.854
R2470.832
SRR1443713GTEX0.829
SRR13130970.825

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