Research Use Only. KIRhub outputs are computational research artifacts. They are not validated for clinical decision-making, diagnosis, or treatment.

Primary targets: CK1EPSILON, PI3K · FDA status: FDA Approval Withdrawn

Selectivity scorecard

MeasuredDerived
KISS
98.74
Gini
0.670
CATDS
0.023

Computed from wild-type kinome inhibition at 1 μM. Gini reproduces the published values within tolerance; KISS and CATDS are computed but pending reconciliation with the paper's reference code.

Polypharmacology radar

MeasuredDerived

Top 20 strongest-inhibited wild-type kinases for Umbralisib. Strongest target: RAF1 at 99.8% inhibition.

Accessible data table
RankTargetInhibition %Residual activity %
1RAF199.8%0.2%
2JAK196.4%3.6%
3JAK296.2%3.8%
4YSK4_MAP3K1995.9%4.1%
5C_KIT94.5%5.5%
6ARAF84.9%15.1%
7DDR279.8%20.2%
8TAOK2_TAO175.4%24.6%
9FMS74.9%25.1%
10ZAK_MLTK71.9%28.1%
11KDR_VEGFR267.8%32.2%
12JAK367.1%32.9%
13HIPK467.0%33.0%
14LYN65.5%34.5%
15BRAF65.3%34.7%
16TYK262.2%37.8%
17LCK61.9%38.1%
18FLT4_VEGFR357.8%42.2%
19ABL2_ARG55.8%44.2%
20CK1EPSILON55.6%44.4%

Selectivity landscape

MeasuredDerived

Where Umbralisib sits in the 92-drug selectivity landscape (KISS vs Gini). The highlighted point is Umbralisib.

Atlas insights for Umbralisib

MeasuredReference

Pathway-space view of what this drug actually does, drawn from the Pathway Atlas.

On-target vs off-target shadow

DerivedMeasured

How much of this drug's pathway perturbation comes from primary targets vs polypharmacology vs 2nd-order propagation. When off-target dominates, the FDA label is the smallest description of the drug.

On-target0%
Off-target100%
Ghost (2nd-order)0%
PathwayCompositionTotal |Π|
ADIPOGENESIS
1763.20
ALLOGRAFT_REJECTION
4950.79
ANDROGEN_RESPONSE
546.73
ANGIOGENESIS
584.31
APICAL_JUNCTION
4681.50
APICAL_SURFACE
538.39
APOPTOSIS
3432.81
BILE_ACID_METABOLISM
432.74
CHOLESTEROL_HOMEOSTASIS
531.33
COAGULATION
579.40
COMPLEMENT
2790.18
DNA_REPAIR
737.85
E2F_TARGETS
2560.85
EPITHELIAL_MESENCHYMAL_TRANSITION
910.82
ESTROGEN_RESPONSE_EARLY
1951.95
ESTROGEN_RESPONSE_LATE
1745.23
FATTY_ACID_METABOLISM
372.57
G2M_CHECKPOINT
2408.55
GLYCOLYSIS
1303.62
HEDGEHOG_SIGNALING
382.38
HEME_METABOLISM
1323.40
HYPOXIA
2099.72
IL2_STAT5_SIGNALING
2067.77
IL6_JAK_STAT3_SIGNALING
4102.41
INFLAMMATORY_RESPONSE
3360.77
INTERFERON_ALPHA_RESPONSE
735.16
INTERFERON_GAMMA_RESPONSE
4216.01
KRAS_SIGNALING_DN
471.61
KRAS_SIGNALING_UP
1935.77
MITOTIC_SPINDLE
2731.14
MTORC1_SIGNALING
1804.75
MYC_TARGETS_V1
1906.85
MYC_TARGETS_V2
499.02
MYOGENESIS
1448.65
NOTCH_SIGNALING
110.14
OXIDATIVE_PHOSPHORYLATION
647.11
P53_PATHWAY
1768.52
PANCREAS_BETA_CELLS
125.77
PEROXISOME
462.23
PI3K_AKT_MTOR_SIGNALING
5104.11
PROTEIN_SECRETION
905.11
REACTIVE_OXYGEN_SPECIES_PATHWAY
200.96
SPERMATOGENESIS
1233.69
TGF_BETA_SIGNALING
913.38
TNFA_SIGNALING_VIA_NFKB
2857.10
UNFOLDED_PROTEIN_RESPONSE
817.99
UV_RESPONSE_DN
2775.34
UV_RESPONSE_UP
1931.49
WNT_BETA_CATENIN_SIGNALING
945.51
XENOBIOTIC_METABOLISM
751.62

See this drug on the perturbation map →

Hallmarks-of-Cancer reach

DerivedReference

Projection onto the 10 canonical Hanahan & Weinberg hallmarks. Breadth = how many hallmarks this drug meaningfully perturbs.

ProliferationEvading apoptosisAngiogenesisInvasion / metastasisReplicative immortalityDeregulated metabolismImmune evasionGenome instabilityInflammationGrowth signaling

Breadth = 3.00 bits (max possible across 10 hallmarks = 3.32 bits). Multi-hallmark agent — broad polypharmacology.

Compare against the full catalog →

Anti-tumor matches — the "ideal patient" search

ModeledDerived

Top 5 real tumors closest to this drug's ideal patient (the tumor whose pathway state = −Π_d). Closest match cosine = 0.836

SampleCancer typecos to ideal
EPT0291EPN0.836
SRR233037520.828
TCGA-CF-A5U8-01A-11R-A28M-070.822
SRR108999840.819
TCGA-FD-A43X-01A-11R-A23W-070.818

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