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Dabrafenib

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Primary targets: BRAF · FDA status: FDA Approved

Selectivity scorecard

KISS
94.74
Gini
0.633
CATDS
0.011

Computed from wild-type kinome inhibition at 1 μM. Gini reproduces the published values within tolerance; KISS and CATDS are computed but pending reconciliation with the paper's reference code.

Polypharmacology radar

Top 20 strongest-inhibited wild-type kinases for Dabrafenib. Strongest target: RAF1 at 99.8% inhibition.

Accessible data table
RankTargetInhibition %Residual activity %
1RAF199.8%0.2%
2NEK999.7%0.3%
3LIMK199.0%1.0%
4LIMK298.9%1.1%
5BRAF98.3%1.7%
6TESK197.0%3.0%
7EIF2AK296.6%3.4%
8ZAK_MLTK96.4%3.6%
9ARAF96.3%3.7%
10RIPK396.0%4.0%
11SIK196.0%4.0%
12DDR295.4%4.6%
13EIF2AK394.5%5.5%
14LCK93.7%6.3%
15PKCMU_PRKD193.3%6.7%
16CSK92.7%7.3%
17TESK292.4%7.6%
18ALK6_BMPR1B92.3%7.7%
19PKCNU_PRKD392.3%7.7%
20NEK1192.1%7.9%

Selectivity landscape

Where Dabrafenib sits in the 92-drug selectivity landscape (KISS vs Gini). The highlighted point is Dabrafenib.

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