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Pralsetinib

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Primary targets: RET · FDA status: FDA Approved

Selectivity scorecard

KISS
93.43
Gini
0.643
CATDS
0.011

Computed from wild-type kinome inhibition at 1 μM. Gini reproduces the published values within tolerance; KISS and CATDS are computed but pending reconciliation with the paper's reference code.

Polypharmacology radar

Top 20 strongest-inhibited wild-type kinases for Pralsetinib. Strongest target: RET at 100.0% inhibition.

Accessible data table
RankTargetInhibition %Residual activity %
1RET100.0%0.0%
2ERBB4_HER499.7%0.3%
3TRKC99.4%0.6%
4TRKB99.4%0.6%
5EGFR99.1%0.9%
6JAK199.1%0.9%
7JAK299.1%0.9%
8TRKA98.7%1.3%
9JAK398.1%1.9%
10FLT397.6%2.4%
11ERBB2_HER297.5%2.5%
12ROS_ROS197.3%2.7%
13DDR196.9%3.1%
14TYK296.8%3.2%
15MLK3_MAP3K1196.5%3.5%
16FLT4_VEGFR396.5%3.5%
17LIMK196.4%3.6%
18BLK96.1%3.9%
19DDR295.9%4.1%
20C_KIT93.8%6.2%

Selectivity landscape

Where Pralsetinib sits in the 92-drug selectivity landscape (KISS vs Gini). The highlighted point is Pralsetinib.

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